Fig. 3From: Mechanisms of Actinidia chinensis Planch in treating colon cancer based on the integration of network pharmacology, molecular docking, and experimental verificationGO functional and KEGG pathway enrichment of ACP target genes (Count numbers ≥ 50). A GO enrichment of ACP targets. GO:0005829, cytosol; GO:0005737, cytoplasm; GO:0005886, plasma membrane; GO:0005634, nucleus; GO:0016021, integral component of membrane; GO:0005654, nucleoplasm; GO:0070062, extracellular exosome; GO:0016020, membrane; GO:0005576, extracellular region; GO:0005887, integral component of plasma membrane; GO:0005615, extracellular space; GO:0005739, mitochondrion; GO:0005783, endoplasmic reticulum; GO:0005789, endoplasmic reticulum membrane; GO:0000785, chromatin; GO:0005515, protein binding; GO:0042802, identical protein binding; GO:0005524, ATP binding; GO:0046872, metal ion binding; GO:0019899, enzyme binding; GO:0042803, protein homodimerization activity; GO:0008270, zinc ion binding; GO:0003677, DNA binding; GO:0007165, signal transduction; GO:0045944, positive regulation of transcription from RNA polymerase II promoter; GO:0045893, positive regulation of transcription, DNA-templated; GO:0008284, positive regulation of cell proliferation; GO:0006468, protein phosphorylation; GO:0042493, response to drug; GO:0010628, positive regulation of gene expression; GO:0043066, negative regulation of apoptotic process; GO:0000122, negative regulation of transcription from RNA polymerase II promoter. B KEGG pathway enrichment analysis of ACP targets. The dots represent the number of genes, and the shades of color represent the logarithm of the base 10 P valueBack to article page