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Table 2 Distribution of SNPs of IDOL gene in Han male subjects

From: Genetic polymorphism of IDOL gene was associated with the susceptibility of coronary artery disease in Han population in Xinjiang, China

Genotype

Model

 

Case (n, %)

Control (n, %)

P value

rs2072783

(A > G)

Codominant

AA

120 (36.0)

55 (33.3)

0.838

GA

157 (47.1)

81 (49.1)

 

GG

56 (16.8)

29 (17.6)

 

Dominant

AA

120 (36.0)

55 (33.3)

0.552

GA + GG

213 (64.0)

110 (66.7)

 

Recessive

AA + GA

277 (83.2)

136 (82.4)

0.832

GG

56 (16.8)

29 (17.6)

 

Additive

GG + AA

176 (52.9)

84 (50.9)

0.683

GA

157 (47.1)

81 (49.1)

 

A

397 (62.4)

191 (57.9)

0.601

G

269 (37.6)

139 (42.1)

 

rs2205796

(T > G)

Codominant

TT

198 (59.5)

104 (63.0)

0.283

GT

124 (37.2)

52 (31.5)

 

GG

11 (3.3)

9 (5.5)

 

Dominant

TT

19,859.5)

104 (63.0)

0.443

GT + GG

135 (40.5)

61 (37.0)

 

Recessive

TT + GT

322 (96.7)

156 (94.5)

0.250

GG

11 (3.3)

9 (5.5)

 

Additive

GG + TT

209 (62.8)

113 (68.5)

0.209

GT

124 (37.2)

52 (31.5)

 

T

520 (78.1)

260 (78.8)

0.798

G

146 (21.9)

70 (21.2)

 

rs909562

(A > G)

Codominant

AA

134 (40.2)

55 (33.3)

0.316

GA

151 (45.3)

82 (49.7)

 

GG

48 (14.4)

28 (17.0)

 

Dominant

AA

134 (40.2)

55 (33.3)

0.135

GA + GG

199 (59.8)

110 (66.7)

 

Recessive

GA + AA

285 (85.6)

137 (83.0)

0.455

GG

48 (14.4)

28 (17.0)

 

Additive

GG + AA

182 (54.7)

83 (50.3)

0.360

GA

151 (45.3)

82 (49.7)

 

A

419 (62.9)

192 (58.2)

0.149

G

247 (37.1)

138 (41.8)

 
  1. Note: In the Han male participants, there were no significant differences in the distributions of genotypes and alleles, dominant model, recessive model, and additive model for three SNPs between case and control groups (all P > 0.05)